Harvard Scientists Discover a New Treatment for Diabetes
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Harvard & Georgia Tech have joined forces with Missouri scientists University to create a new diabetes treatment. The procedure involves the transplantation of insulin-producing pancreatic cells.
An estimated 1.7 million Americans have type 1 diabetes. Type 1 diabetes may develop during childhood. It can also develop later in life.
There is no cure for type 1 Diabetes. You can treat type 1 diabetes by taking insulin, maintaining your diet, exercising regularly, and managing sugar levels. Scientists also found a new method of treating promising.
Researchers from Harvard University, the Institute of Georgia, the University of Missouri & Georgia Institute of Technology have demonstrated that novel Type 1 treatment can be used in large-animal models of diabetes. This was available in Science Advance on May 13th. They transferred insulin-producing pancreas cells from donors to recipients using a method known as pancreatic islets.
Haval Shirwan, a professor of pediatrics and immunology & molecular biology at the MU School of Medicine, says that Type 1 diabetes patients may experience an immune system malfunction, leading them to target themselves.
Shirwan stated that the immune system manages a defense mechanism that protects individuals from infections. Type 1 diabetes is caused by the immune system misidentifying insulin-producing cells in your pancreas and destroying them. Usually, once an apparent threat or danger is removed, the immune system’s command & control mechanism kicks into action to remove any rogue cell. Type 1 diabetes could occur if this mechanism fails.
It disturbs the body’s capacity to produce and use insulin, a hormone that regulates blood sugar metabolism. Type 1 diabetes cannot control blood sugar levels as they don’t produce insulin. This can cause kidney damage, heart disease, & vision loss.
Shirwan & Esma, a professor of molecular immunology and microbiology & child health at MU School of Medicine, have spent two decades focusing on an apoptosis mechanism that stops “rogue immune cells” from producing diabetes and rejecting transplanted pancreatic islets. They attached a molecule known as FasL onto the islets’ surfaces.
Yolcu, one of the study’s first authors, stated that a molecule known as FasL interrelates with another molecule known as Fas on rogue immune cells. This is the reason for them to die. Our team created a treatment that allowed the making of a novel FasL form and presented it on transplanted pancreatic islet cells to avoid rejection by rogue cells. Rogue cells that are insulin-producing pancreatic
With over 4 years of experience in the research industry, Kathleen is generally engrossed in market consulting projects, catering primarily to domains such as ICT, Health & Pharma, and packaging. She is highly proficient in managing both B2C and B2B projects, with an emphasis on consumer preference analysis, key executive interviews, etc. When Kathleen isn’t deconstructing market performance trajectories, she can be found hanging out with her pet cat ‘Sniffles’.
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